SDS

Date SDS Prepared: May 31, 2015
Page 4 of 5
9.
PHYSICAL AND CHEMICAL PROPERTIES
Appearance: Brittle solid; color may vary
Odor: Odorless
Melting Point:
Not Available (>2200
0
F)
Boiling Point: Not applicable
Vapor Pressure: Not applicable
Vapor Density (Air = 1): Not applicable
Solubility in Water: Insoluble
Specific Gravity (H2) = 1): 1.6 to 2.1
Percent Volatile by Volume: Not applicable
Evaporation Rate (Ethyl Ether = 1):
Not applicable
Viscosity: Not applicable
Volatility: 0 g/L Volatile Organic Compounds (VOCs)
10. STABILITY AND REACTIVITY
Stability: Stable in current form.
Conditions to Avoid: Avoid contact with acids (e.g., acetic, hydrofluoric, etc.)
Incompatibility (Materials to Avoid): Avoid contact with acids (e.g., acetic, hydrofluoric, etc.)
Hazardous Polymerization: Will not occur.
Hazardous Decomposition Products: None.
11. TOXICOLOGICAL INFORMATION
Potential Health Effects
Primary Routes of Exposure
Acute Effects
Chronic Effects
Potential Adverse Interactions
Carcinogen Status
Overview of Animal Testing
Oral (silica) Lethality
LD50 Rat oral >22,500 mg/kg
LD50 Mouse oral >15,000 mg/kg
LC50 Carp >10,000 mg/l (per 72 hr.)
No acute effects from exposure to intact tile are known. Working with broken or cut tile produces a potential for cuts to the hands and
exposed body parts. Acute effects such as eye irritation may occur if associated with high dust operations such as dry cutting tile or
during the removal of installed tile. In very rare cases, symptoms of acute silicosis, a form of silicosis (a nodular pulmonary fibrosis)
associated with exposure to respirable crystalline silica, may develop following acute exposure to extremely dusty environments caused by
generation of tile dust. Signs such as labored breathing and early fatigue may indicate silicosis; however, these same symptoms can arise
from many other causes.
No chronic effects are known for exposure to intact tile. Long-term, continual exposure to respirable crystalline silica at or above
established permissible occupational exposure limits may lead to the development of silicosis, a nodular pulmonary fibrosis (NPF). NPFs
are also associated with pulmonary tuberculosis, bronchitis, emphysema, and other airway diseases. This type of chronic exposure to
silica dust may also result in the development of autoimmune disorders, chronic renal disease, and other adverse health effects.
Recent epidemiologic studies demonstrate that workers exposed to elevated silica concentrations have a significant risk of developing
chronic silicosis. Signs such as labored breathing and early fatigue may indicate silicosis; however, these same symptoms can also arise
from many other causes.
Silicosis may be complicated by severe mycobacterial or fungal infections and result in tuberculosis (TB). Epidemiologic studies have
established that silicosis is a risk factor for developing TB. Any existing respiratory or pulmonary diseases may be complicated by
exposure to respirable crystalline silica. Smoking may increase the risk of adverse effects if done in conjunction with occupational
exposure to silica dust at or above permissible exposure limits.
Respirable crystalline silica is classified by the International Agency for Research on Cancer (IRAC) as a Group I Carcinogen
(carcinogenic to humans). The National Toxicology Program (9th Report) lists respirable crystalline silica as “Known to be a Human
Carcinogen”. USDOL/OSHA and NIOSH have recommended that crystalline silica be considered a potential occupational carcinogen.
Short term experimental studies of rats have found that intratracheal instillation of quartz particles leads to the formation of discrete
silicotic nodules in rats, mice and hamsters.
None for intact tile. Inhalation and potential exposure to eyes, hands, or other body parts if contact is made with broken tile, and/or
during procedures involving the cutting of tiles, and/or for operations involving the removal of installed tiles.