SDS

SAFETY DATA SHEET
Compare-N-Save® Systemic Tree & Shrub
Insect Drench
August 26, 2019 Page 4 of 7
10. STABILITY AND REACTIVITY
Reactivity: Not reactive.
Chemical Stability: This material is stable under normal handling and storage conditions.
Possibility of Hazardous Reactions: Will not occur.
Conditions to Avoid: Excessive heat. For imidacloprid, strong exothermal reaction above 200 C.
Incompatible Materials: Not known.
Hazardous Decomposition Products: Under fire conditions, may produce gases such as hydrogen chloride,
hydrogen cyanide, and oxides of carbon and nitrogen.
11. TOXICOLOGICAL INFORMATION
Likely Routes of Exposure: Inhalation, eye and skin contact.
Symptoms of Exposure: Mildly irritating to the eyes based on toxicity studies. Minimally toxic and slightly irritating
to the skin based on toxicity studies. Slightly toxic if ingested based on toxicity studies. Low inhalation toxicity.
Delayed, immediate and chronic effects of exposure: None known
Toxicological Data:
Data from laboratory studies conducted are summarized below:
Oral: Rat LD
50
: > 5,000 mg/kg (female)
Dermal: Rat LD
50
: >5,000 mg/kg
Inhalation: Rat 4-hr LC
50
: >2.02 mg/L
Eye Irritation: Rabbit: Mildly irritating (cleared within 72 hrs)
Skin Irritation: Rabbit: Slightly irritating
Skin Sensitization: Not a contact sensitizer in guinea pigs following repeated skin exposure.
Subchronic (Target Organ) Effects: Repeated overexposure to imidacloprid, may effect heart, thyroid, blood
chemistry, and liver. Ingestion of large amounts of propylene glycol has resulted in symptoms of reversible central
nervous system depression including stupor, rapid breathing and heartbeat, profuse sweating and seizures.
Carcinogenicity / Chronic Health Effects: Prolonged overexposure to imidacloprid can cause effects to the
thyroid. Repeated overexposure to propylene glycol can produce central nervous system depression, hemolysis
and minimal kidney damage. Imidacloprid did not cause cancer in laboratory animal studies. The U.S. EPA has
given imidacloprid a Group E classification (evidence of non-carcinogenicity in humans).
Reproductive Toxicity: In a two-generation reproduction study in rats, imidacloprid produced reduced mean body
weights and body weight gains. No other reproductive effects were observed.
Developmental Toxicity: Rat and rabbit studies on imidacloprid resulted in skeletal abnormalities, increased
resorptions (rabbits) and reduced body weight gains at doses that were also toxic to mother animals.
Genotoxicity: The imidacloprid mutagenicity studies, taken collectively, demonstrate that imidacloprid is not
genotoxic or mutagenic.
Assessment Carcinogenicity: None listed with ACGIH, IARC, NTP or OSHA.
12. ECOLOGICAL INFORMATION
Environmental Hazards:
This product is toxic to aquatic invertebrates.
Ecotoxicity:
Data on Imidacloprid Technical:
96-hour LC
50
Rainbow Trout: 211 mg/l Japanese Quail Oral LD
50
: 31 mg/kg
48-hour EC
50
Daphnia: 85 mg/l Bobwhite Quail Oral LD
50
: 152 mg/kg
96-hour LC
50
Mysid: 0.038 ppm House Sparrow Oral LD
50
: 41 mg/kg
48-hour Honey Bee Contact LD
50
: 0.078 g/bee 48-hour Honey Bee Oral LD
50
: 0.0039 g/bee
Environmental Fate:
Hydrolysis half-life of imidacloprid is greater than 30 days at pH 7 and 25C. The aqueous photolysis half-life is
less than 3 hours. The soil surface photolysis of imidacloprid has a half-life of 39 days, and in soil, the half-life
ranged from 26 to 229 days.