SDS

SBM LIFE SCIENCE CORP.
SAFETY DATA SHEET
BIOADVANCED SCIENCE-BASED
SOLUTIONS 5-IN-1 WEED & FEED
Version 1.0 / USA
6/10
Revision Date: 03/8/2020
Ingestion:
Inhalation:
Product Acute Toxicity Values: LD50 oral rat >2000 mg/kg, LD50 dermal rat >2000 mg/kg; LC50
Inhalation of dust may cause irritation of the mucous membranes and
upper respiratory tract
Swallowing may cause gastrointestinal distress with nausea and
diarrhea and nervous system effects.
Chronic Effects:
Sensitization:
Germ Cell Mutagenicity:
Carcinogenicity:
OSHA:
Reproductive/
Developmental Toxicity:
Additional Information:
inhalation rat >2.12 mg/L (no mortalities at maximum concentration)
Product did not cause sensitization in the LLNA test.
Repeated overexposure to phenoxy herbicides may affect the liver,
kidneys, blood chemistry, and gross motor function. Rare cases of
peripheral nerve damage have been reported, but extensive animal
studies have failed to substantiate these observations, even at high
doses for prolonged periods. Repeated exposure to dicamba may
cause liver changes or a decrease in body weight. Repeated
exposure to quinclorac may affect the kidneys, liver and blood.
Components are not germ cell mutagens. There have been some
positive and some negative studies, but the weight of evidence is
that 2,4-D is not mutagenic. In vitro and in vivo tests with dicamba
did not demonstrate mutagenic effects. Quinclorac was not
mutagenic in various studies.
The International Agency for Research on Cancer (IARC) lists 2,4-D
and exposure to chlorophenoxy herbicides as a class 2B carcinogen
(see Monograph 113, 2016) – inadequate evidence in humans and
limited evidence in laboratory animals. The U.S. EPA has given 2,4-
D a Class D classification (not classifiable as to human
carcinogenicity). Dicamba did not cause cancer in long-term animal
studies. The U.S. EPA has given dicamba a Class D classification
(not classifiable as to human carcinogenicity). Quinclorac was
negative for carcinogenicity in laboratory animals.
None.
Ingredients are not reproductive toxins. No adverse reproductive
effects have been observed in laboratory animal studies with 2,4-
D. Studies in laboratory animals with 2,4-D have shown
decreased fetal body weights and delayed development in the
offspring at maternally toxic doses. Dicamba did not interfere with
fertility in reproduction studies or cause developmental effects in
laboratory animals. Quinclorac did not cause developmental
toxicity in rats but caused adverse effects in rabbits at maternally
toxic doses.
Only acute toxicity studies have been performed on the formulated
product. The non-acute information pertains to the active
ingredient(s).