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Exposu
re time: 1 h
Determined in the form of a respirable aerosol.
Extrapolated from the 4 hr LC50.
(actual)
Acute dermal toxicity
LD50 (male/female combined rat) > 4,000 mg/kg
Skin irritation
No skin irritation (rabbit)
Eye irritation
No eye irritation (rabbit)
Sensitisation
Non-sensitizing. (guinea pig)
Assessment repeated dose toxicity
Imidacloprid did not cause specific target organ toxicity in experimental animal studies.
Tau-fluvalinate did not cause specific target organ toxicity in experimental animal studies.
Tebuconazole did not cause specific target organ toxicity in experimental animal studies.
Assessment mutagenicity
Imidacloprid was not mutagenic or genotoxic based on the overall weight of evidence in a battery of in
vitro and in vivo tests.
Tau-fluvalinate was not mutagenic or genotoxic in a battery of in vitro and in vivo tests.
Tebuconazole was not mutagenic or genotoxic in a battery of in vitro and in vivo tests.
Assessment carcinogenicity
Imidacloprid was not carcinogenic in lifetime feeding studies in rats and mice.
Tau-fluvalinate was not carcinogenic in lifetime feeding studies in rats and mice.
Tebuconazole caused at high dose levels an increased incidence of tumours in mice in the following
organ(s): liver. The mechanism of tumour formation is not considered to be relevant to man.
ACGIH
1-Propanol 71-23-8 Group A4
NTP
None.
IARC
None.
OSHA
None.
Assessment toxicity to reproduction
Imidacloprid caused reproduction toxicity in a two-generation study in rats only at dose levels also toxic
to the parent animals. The reproduction toxicity seen with Imidacloprid is related to parental toxicity.
Tau-fluvalinate did not cause reproductive toxicity in a two-generation study in rats.
Tebuconazole caused reproduction toxicity in a two-generation study in rats only at dose levels also
toxic to the parent animals. The reproduction toxicity seen with Tebuconazole is related to parental
toxicity.
Assessment developmental toxicity
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Revision Date: 01/31/2019