MSDS
Bayer Environmental Science
Material Safety Data Sheet
MSDS Number: 102000012177
BAYER ADVANCED COMPLETE BRAND INSECT KILLER
FOR SOIL & TURF CONCENTRATE
MSDS Version 2.0
Page 5 of 8
In a subchronic dietary study in rats treated with beta-cyfluthrin, effects included
reduced body weight gains and feed consumption, uncoordinated gait, and skin
injuries of the neck and head from excessive preening due to the local irritant
effect of the test material.
In a 4-week inhalation study in rats exposed to beta-cyfluthrin, effects observed
included ungroomed fur, piloerection, hyper- and hypoactivity, reduced body
weight gains, hematological changes and reduced organ weights (thymus,
spleen).
IMIDACLOPRID TECHNICAL
In a 3-week dermal toxicity study, rabbits treated with imidacloprid showed no
local or systemic effects at levels up to and including 1000 mg/kg, the limit dose.
In a 4-week inhalation study, rats exposed to high concentrations of imidacloprid
exhibited decreased body weight gains and changes in clinical chemistries and
organ weights.
Chronic Toxicity
IMIDACLOPRID TECHNICAL
In chronic dietary studies in rats and dogs exposed to imidacloprid, the target
organs were the thyroids and/or liver.
Assessment Carcinogenicity
IMIDACLOPRID TECHNICAL
In oncogenicity studies in rats and mice, imidacloprid was not considered carcinogenic in either species.
ACGIH
None.
NTP
None.
IARC
None.
OSHA
None.
Reproductive &
Developmental Toxicity
BETA-CYFLUTHRIN TECHNICAL
DEVELOPMENTAL TOXICITY: In an oral developmental toxicity study in rats
treated with beta cyfluthrin, decreased fetal body weights and an increased
incidence of skeletal findings were observed at the maternally toxic and lethal
high dose level (40 mg/kg).
IMIDACLOPRID TECHNICAL
DEVELOPMENTAL TOXICITY: In developmental toxicity studies in rats and
rabbits, there was no evidence of an embryotoxic or teratogenic potential for
imidacloprid. In both species, developmental effects were observed only at high
doses and in conjunction with maternal toxicity.
REPRODUCTION: In a two-generation reproduction study in rats, imidacloprid
was not a primary reproductive toxicant. Offspring exhibited reduced body